Preliminary studies show conolidine may well inhibit certain ion channels, lowering neuronal excitability and restricting pain alerts. This mechanism is particularly related in neuropathic discomfort, where abnormal signaling results in persistent distress. In addition, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to soreness notion. Computational d... https://samuelb121ozq1.wikiexcerpt.com/user